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Activated glial cells interfere with monoaminergic neurotransmission by reducing serotonin synthesis, altering dopamine signaling, and impairing norepinephrine regulation, thereby directly contributing to mood dysregulation and anhedonia (31)

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At this time, mold IgE and IgG testing were performed to try to ascertain whether the persistent symptoms were exacerbated by consistent exposure, confirming significant immune sensitization

Similarly, the bioenergetic deficit driven by mtDNA dysfunction is a fundamental driver of T cell exhaustion